Circulating mitochondria promoted endothelial cGAS-derived neuroinflammation in subfornical organ to aggravate sympathetic overdrive in heart failure mice, Journal of Neuroinflammation

Neuroimmune cardiovascular interfaces control atherosclerosis

Neurohormonal activation in heart failure with reduced ejection fraction

Intercellular mitochondrial component transfer triggers ischemic cardiac fibrosis - ScienceDirect

Mitochondrial-derived peptides in cardiovascular disease: Novel insights and therapeutic opportunities - ScienceDirect

Sorting mechanism and biological effect of mitoEVs. (a). Possible

The experimental flow chart of the study.

Circulating mitochondria promoted endothelial cGAS-derived neuroinflammation in subfornical organ to aggravate sympathetic overdrive in heart failure mice, Journal of Neuroinflammation

Inhibition of cGAS-STING by JQ1 alleviates oxidative stress-induced retina inflammation and degeneration

Circulating mitochondria promoted endothelial cGAS-derived neuroinflammation in subfornical organ to aggravate sympathetic overdrive in heart failure mice, Journal of Neuroinflammation

Ultrasound-responsive low-dose doxorubicin liposomes trigger mitochondrial DNA release and activate cGAS-STING-mediated antitumour immunity

Ultrasound-responsive low-dose doxorubicin liposomes trigger mitochondrial DNA release and activate cGAS-STING-mediated antitumour immunity

Circulating mitochondria promoted endothelial cGAS-derived neuroinflammation in subfornical organ to aggravate sympathetic overdrive in heart failure mice, Journal of Neuroinflammation

Circulating mitochondria promoted endothelial cGAS-derived neuroinflammation in subfornical organ to aggravate sympathetic overdrive in heart failure mice, Journal of Neuroinflammation

Decreased MFN2 activates the cGAS-STING pathway in diabetic myocardial ischaemia–reperfusion by triggering the release of mitochondrial DNA, Cell Communication and Signaling